Diana Prata
Gabinete no IBEB
0.07
Contactos
E-mail: diana.prata[at]fc.ul.pt
Redes profissionais
Tópicos de investigação
- Neuroscience
- Oxytocin
- Dopamine
- Empathy
- Cooperation
- Trust
- Social cognition
- Neuroimaging
- Eye-tracking
- Pupillometry
- Electroencephalography
- Skin conductance response
- Electrocardiography
- Schizophrenia
- Autism
- Dance
- Bonding
- Dementia
- Neurodegeneration
- Artificial intelligence
- Machine learning
- Personalized medicine
- Biomarkers
- Psychiatry
- Neurology
- Psychology
Biografia
Sou uma Marie Curie alumnae, Líder de Grupo e Professora Assistente na Universidade de Lisboa desde 2015 (depois de 12 anos no Instituto de Psiquiatria, Psicologia e Neurociência do King’s College London; IoPPN – KCL) onde fundei um grupo de investigação em Biomedical Neuroscience lab (dpratalab.wordpress.com), bem como Docente Convidada do IoPPN-KCL e Colaboradora do Centro de Investigação Psicológica e Intervenção Social (CIS) do IUL-ISCTE. Fui distinguida com o 3º Prémio Marie Curie 2017 como ‘Most Promising Scientist in Innovation & Entrepreneurship’; três prémios baseados em publicações por 2 universidades (os Prémios Científicos ISCTE-IUL 2019 e 2020 e uma Menção Honrosa 2020 pela Univ. Lisboa). Em 2023, recebi um contrato de Emprego Científico de 2022, classificado em 1º lugar em Psicologia em todo o país.
Tornei-me bióloga na Universidade de Lisboa em 2002, e mudei-me para o Instituto de Psiquiatria, Psicologia e Neurociências, do KCL, como bolseira Leonardo da Vinci, apaixonada pela neurociência humana. Lá, comecei a trabalhar na genética da doença mental. Com uma bolsa da FCT, aproximei-me “mais do cérebro” e concluí aí o doutoramento em genética de neuroimagiologia em 2009. Dois anos depois, recebi a prestigiada bolsa de pós-doutoramento NIHR do Reino Unido para trabalhar num biomarcador multimodal para psicose, depois uma posição de Lectureship na KCL, também uma promoção KCL Academic Advancement, e fui distinguida pela KCL como Top Early Career Researcher no UK Research Excellence Framework 2014.
Em termos de investigação, enquanto estava no Reino Unido, publiquei a primeira evidência de que os genes de risco para esquizofrenia podem predispor ao transtorno bipolar. Durante meu doutorado, fui pioneira na combinação de genética e neuroimagem para elucidar como as mutações genéticas implicadas nessas doenças afetam a estrutura da substância branca e a função cerebral. Como pós-doutoranda, descrevi como algumas dessas mutações influenciam a resposta aos antipsicóticos; e iniciei um estudo genético/imagem/ambiental para prever o início da psicose, usando aprendizagem automática. Em 2015, fundei o nosso laboratório de investigação em Portugal, tendo-me sido atribuído um contrato de investigador FCT a tempo inteiro e sem vínculo.
Como Investigadora Principal, em Portugal, montei uma instalação psicofisiológica multimodal única (MRI, EEG, SCR, ECG & eye-tracking) que permite a uma equipa diversificada realizar neurociência humana interdisciplinar avançada: médicos, psiquiatras, biólogos, psicólogos , antropólogo e engenheiros. Tentamos desvendar os mecanismos do ainda pouco estudado sistema de oxitocina – usando farmacologia, neuroimagiologia, psicofisiologia, genética e modelagem estatística avançada – para entender melhor como os humanos desenvolveram habilidades sociais e encontrar pistas sobre como melhorá-las no autismo, psicopatia e psicose. Além disso, desenvolvemos biomarcadores diagnósticos e prognósticos para doenças neurodegenerativas. A nossa investigação cresceu vertiginosamente na variedade de técnicas que usamos, colaborações, mídia e especialistas e estudantes que atraímos.
No geral, a missão do nosso laboratório é: 1) usando a neurociência, destacar a importância da dimensão social de nossas vidas para o nosso bem-estar pessoal e social e melhorá-la na saúde e na doença; e 2) conseguir isso em um ambiente de laboratório empático, criativo, centrado no trabalho em equipa e socialmente saudável.
[informação adicional e atualizada em https://dpratalab.wordpress.com/]
Mais informação em https://en.wikipedia.org/wiki/Diana_Prata
Publicações
Publicações em revistas
(2024) Oxytocin modulates neural activity during early perceptual salience attribution, Psychoneuroendocrinology 161, p. 106950, Pergamon, doi:10.1016/j.psyneuen.2023.106950
(2024) The European Portuguese Version of the Brief Negative Symptom Scale, Psychopathology 57(1), p. 76-80, S. Karger AG
(2023) Psychosis Endophenotypes: A Gene-Set-Specific Polygenic Risk Score Analysis, Schizophrenia Bulletin 49(6), p. 1625-1636, Oxford University Press US, doi:10.1093/schbul/sbad088
(2023) Oxytocin modulates neural activity during early perceptual salience attribution, Psychoneuroendocrinology 161, p. 106950, Pergamon, doi:10.1016/j.psyneuen.2023.106950
(2023) Prediction of transition to psychosis from an at-risk mental state using structural neuroimaging, genetic, and environmental data, Frontiers in Psychiatry 13, p. 3090, Frontiers, doi:10.3389/fpsyt.2022.1086038
(2023) Computer anthropomorphisation in a socio-economic dilemma, Behavior Research Methods, p. 1-13, Springer US New York, doi:10.3758/s13428-023-02071-y
(2023) Temporal profile of intranasal oxytocin in the human autonomic nervous system at rest: An electrocardiography and pupillometry study, Journal of Psychopharmacology 37(6), p. 566-576, SAGE Publications Sage UK: London, England, doi:10.1177/02698811231158233
(2023) The European Portuguese Version of the Brief Negative Symptom Scale, Psychopathology 57(1), p. 1-1, S. Karger AG, doi:10.1159/000530705
(2022) Cultural differences in vocal emotion recognition: a behavioural and skin conductance study in Portugal and Guinea-Bissau, Psychological Research 86(2), p. 597-616, Springer, doi:10.1007/s00426-021-01498-2
(2022) Sex Differences in Functional Connectivity Between Resting State Brain Networks in Autism Spectrum Disorder, Journal of Autism and Developmental Disorders 52(7), p. 3088-3101, Springer US, doi:10.1007/s10803-021-05191-6
(2022) Temporal dynamics of intranasal oxytocin in human brain electrophysiology, Cerebral Cortex 32(14), p. 3110-3126, Oxford University Press, doi:10.1093/cercor/bhab404
(2022) Early diagnosis of Alzheimer’s disease using machine learning: a multi-diagnostic, generalizable approach, Alzheimer's Research and Therapy 14(1), p. 107, BioMed Central London, doi:10.1186/s13195-022-01047-y
(2022) Effects of psychosis-associated genetic markers on brain volumetry: a systematic review of replicated findings and an independent validation, Psychological Medicine 52(16), p. 3753-3768, Cambridge University Press, doi:10.1017/S0033291722002896
(2022) Neuroimaging genetics of oxytocin: A transcriptomics-informed systematic review, Neuroscience and Biobehavioral Reviews 142, p. 104912, Elsevier, doi:10.1016/j.neubiorev.2022.104912
(2022) Cultural differences in vocal emotion recognition: a behavioural and skin conductance study in Portugal and Guinea-Bissau, Psychological Research 86(2), p. 597-616, Springer Berlin Heidelberg, doi:10.1007/s00426-021-01498-2
(2021) Pupil dilation reflects the authenticity of received nonverbal vocalizations, Scientific Reports 11(1), p. 3733, Nature Publishing Group UK London, doi:10.1038/s41598-021-83070-x
(2021) Evaluation of genotype-based gene expression model performance: a cross-framework and cross-dataset study, Genes 12(10), p. 1531, MDPI
(2021) The neural basis of authenticity recognition in laughter and crying, Scientific Reports 11(1), p. 23750, Nature Publishing Group UK London, doi:10.1038/s41598-021-03131-z
(2021) Epigenetic mediation of akt1 rs1130233′s effect on delta-9-tetrahydrocannabinol-induced medial temporal function during fear processing, Brain Sciences 11(9), p. 1240, MDPI, doi:10.3390/brainsci11091240
(2021) Pupil dilation reflects the authenticity of received nonverbal vocalizations, Scientific Reports 11(1), p. 3733, Nature Publishing Group UK London, doi:10.1038/s41598-021-83070-x
(2021) Genetic copy number variants, cognition and psychosis: a meta-analysis and a family study, Molecular Psychiatry 26(9), p. 5307-5319, Nature Publishing Group UK London, doi:10.1038/s41380-020-0820-7
(2020) Oxytocin and vasopressin modulation of prisoner’s dilemma strategies, Journal of Psychopharmacology 34(8), p. 891-900, Sage Publications Sage UK: London, England, doi:10.1177/0269881120913145
(2020) Comparing SPM12 and CAT12 segmentation pipelines: a brain tissue volume-based age and Alzheimer's disease study, Journal of Neuroscience Methods 334, p. 108565, Elsevier, doi:10.1016/j.jneumeth.2019.108565
(2020) Schizophrenia polygenic risk score influence on white matter microstructure, Journal of Psychiatric Research 121, p. 62-67, Pergamon, doi:10.1016/j.jpsychires.2019.11.011
(2019) The effect of psychosis associated CACNA1C, and its epistasis with ZNF804A, on brain function, Genes, Brain and Behavior 18(4), p. e12510, Blackwell Publishing Ltd Oxford, UK
(2019) Unravelling the genetic basis of schizophrenia and bipolar disorder with GWAS: A systematic review, Journal of Psychiatric Research 114, p. 178-207, Elsevier, doi:10.1016/j.jpsychires.2019.04.007
(2019) The effect of psychosis associated CACNA1C, and its epistasis with ZNF804A, on brain function, Genes, Brain and Behavior 18(4), p. e12510, Blackwell Publishing Ltd Oxford, UK, doi:10.1111/gbb.12510
(2018) The impact of psychosis genome-wide associated ZNF804A variation on verbal fluency connectivity, Journal of Psychiatric Research 98, p. 17-21, Pergamon
(2018) The effect of the DISC1 Ser704Cys polymorphism on striatal dopamine synthesis capacity: an [18F]-DOPA PET study, Human molecular genetics 27(20), p. 3498-3506, Oxford University Press
(2018) The effect of the DISC1 Ser704Cys polymorphism on striatal dopamine synthesis capacity: An [18F]-DOPA PET study, Human Molecular Genetics 27(20), p. 3498-3506, Oxford University Press, doi:10.1093/hmg/ddy242
(2018) A polygenic risk score analysis of psychosis endophenotypes across brain functional, structural, and cognitive domains, American Journal of Medical Genetics, Part B: Neuropsychiatric Genetics 177(1), p. 21-34, doi:10.1002/ajmg.b.32581
(2018) How do hypothalamic nonapeptides shape youth's sociality? A systematic review on oxytocin, vasopressin and human socio-emotional development, Neuroscience and Biobehavioral Reviews 90, p. 309-331, Pergamon, doi:10.1016/j.neubiorev.2018.05.004
(2018) The impact of psychosis genome-wide associated ZNF804A variation on verbal fluency connectivity, Journal of Psychiatric Research 98, p. 17-21, Pergamon, doi:10.1016/j.jpsychires.2017.12.005
(2017) The impact of CACNA1C gene, and its epistasis with ZNF804A, on white matter microstructure in health, schizophrenia and bipolar disorder1, Genes, Brain and Behavior 16(4), p. 479-488, Blackwell Publishing Ltd Oxford, UK
(2017) The “highs and lows” of the human brain on dopaminergics: Evidence from neuropharmacology, Neuroscience & Biobehavioral Reviews 80, p. 351-371, Pergamon
(2017) An Examination of Polygenic Score Risk Prediction in Individuals With First-Episode Psychosis, Biological Psychiatry 81(6), p. 470-477, Elsevier, doi:10.1016/j.biopsych.2016.06.028
(2017) The impact of CACNA1C gene, and its epistasis with ZNF804A, on white matter microstructure in health, schizophrenia and bipolar disorder, Genes, Brain and Behavior 16(4), p. 479-488, Blackwell Publishing Ltd Oxford, UK, doi:10.1111/gbb.12355
(2017) The “highs and lows” of the human brain on dopaminergics: Evidence from neuropharmacology, Neuroscience and Biobehavioral Reviews 80, p. 351-371, Pergamon, doi:10.1016/j.neubiorev.2017.06.003
(2016) Genome-wide discovered psychosis-risk gene ZNF804A impacts on white matter microstructure in health, schizophrenia and bipolar disorder, PeerJ 4, p. e1570, PeerJ Inc.
(2016) "Shedding light on a dark question”: Peripheral oxytocin signalling and neurobehavioral responses to intranasal oxytocin in humans, Psychoneuroendocrinology. Commentary. 1580, p. 69-77
(2016) “Shedding light on a dark question”: Peripheral oxytocin signalling and neurobehavioral responses to intranasal oxytocin in humans, Psychoneuroendocrinology 73, p. 271-272, doi:10.1016/j.psyneuen.2016.08.008
(2016) Differences in cannabis-related experiences between patients with a first episode of psychosis and controls, Psychological Medicine 46(5), p. 995-1003, Cambridge University Press, doi:10.1017/S0033291715002494
(2016) Genome-wide discovered psychosis-risk gene ZNF804A impacts on white matter microstructure in health, schizophrenia and bipolar disorder, PeerJ 2016(2), p. e1570, PeerJ Inc., doi:10.7717/peerj.1570
(2015) Predicting clinical response in people at ultra-high risk of psychosis: a systematic and quantitative review, Drug discovery today 20(8), p. 924-927, Elsevier Current Trends
(2015) What is the impact of genome-wide supported risk variants for schizophrenia and bipolar disorder on brain structure and function? A systematic review, Psychological Medicine 45(12), p. 2461-2480, Cambridge University Press, doi:10.1017/S0033291715000537
(2015) Investigação das bases biológicas da cognição na esquizofrenia com Imagiologia Genética, Reabilitação psicossocial e inclusão na saúde mental: da Biologia à Economia da saúde: da inserção à criação artística, p. 179-216, Imprensa da Universidade de Coimbra, doi:10.14195/978-989-26-0852-5_8
(2015) Predicting clinical response in people at ultra-high risk of psychosis: a systematic and quantitative review, Drug Discovery Today 20(8), p. 924-927, Elsevier Current Trends, doi:10.1016/j.drudis.2015.03.003
(2015) As bases biológicas da Esquizofrenia, Reabilitação psicossocial e inclusão na saúde mental: da Biologia à Economia da saúde: da inserção à criação artística, p. 15-49, Imprensa da Universidade de Coimbra, doi:10.14195/978-989-26-0852-5_1
(2014) Role of environmental confounding in the association between FKBP5 and first-episode psychosis, Frontiers in psychiatry 5, p. 84, Frontiers Media SA
(2014) Imagiologia genética, Reabilitação psicossocial e inclusão na saúde mental: da Biologia à Economia da saúde: da inserção à criação art{\'\i}stica, p. 179, Imprensa da Universidade de Coimbra/Coimbra University Press
(2014) Daily use, especially of high-potency cannabis, drives the earlier onset of psychosis in cannabis users, Schizophrenia bulletin 40(6), p. 1509-1517, Oxford University Press US
(2014) Clinically meaningful biomarkers for psychosis: a systematic and quantitative review, Neuroscience & Biobehavioral Reviews 45, p. 134-141, Pergamon
(2014) Investigação das bases biológicas da cognição na esquizofrenia com Imagiologia Genética, Parte: http://hdl. handle. net/10316.2/35952, Imprensa da Universidade de Coimbra
(2014) As bases biológicas da esquizofrenia, Parte: http://hdl. handle. net/10316.2/35952, Imprensa da Universidade de Coimbra
(2014) Protein kinase B (AKT1) genotype mediates sensitivity to cannabis-induced impairments in psychomotor control, Psychological Medicine 44(15), p. 3315-3328, Cambridge University Press, doi:10.1017/S0033291714000920
(2014) Imagiologia genética, Reabilitação psicossocial e inclusão na saúde mental: da Biologia à Economia da saúde: da inserção à criação art{\'\i}stica, p. 179, Imprensa da Universidade de Coimbra/Coimbra University Press
(2014) Role of Environmental Confounding in the Association between FKBP5 and First-Episode Psychosis, Frontiers in Psychiatry 5, p. 84, Frontiers Media SA, doi:10.3389/fpsyt.2014.00084
(2014) Daily use, especially of high-potency cannabis, drives the earlier onset of psychosis in cannabis users, Schizophrenia Bulletin 40(6), p. 1509-1517, Oxford University Press US, doi:10.1093/schbul/sbt181
(2014) Clinically meaningful biomarkers for psychosis: A systematic and quantitative review, Neuroscience and Biobehavioral Reviews 45, p. 134-141, Pergamon, doi:10.1016/j.neubiorev.2014.05.010
(2013) The impact of CACNA1C allelic variation on effective connectivity during emotional processing in bipolar disorder, Molecular psychiatry 18(5), p. 526-527, Nature Publishing Group
(2013) Using genetic, cognitive and multi-modal neuroimaging data to identify ultra-high-risk and first-episode psychosis at the individual level, Psychological medicine 43(12), p. 2547-2562, Cambridge University Press
(2013) Interaction between effects of genes coding for dopamine and glutamate transmission on striatal and parahippocampal function, Human brain mapping 34(9), p. 2244-2258
(2013) Molecular genetic gene-environment studies using candidate genes in schizophrenia: A systematic review, Schizophrenia Research 150(2-3), p. 356-365, Elsevier, doi:10.1016/j.schres.2013.09.010
(2013) Risk variant of oligodendrocyte lineage transcription factor 2 is associated with reduced white matter integrity, Human Brain Mapping 34(9), p. 2025-2031, doi:10.1002/hbm.22045
(2013) The impact of CACNA1C allelic variation on effective connectivity during emotional processing in bipolar disorder, Molecular Psychiatry 18(5), p. 526-527, Nature Publishing Group, doi:10.1038/mp.2012.61
(2013) Using genetic, cognitive and multi-modal neuroimaging data to identify ultra-high-risk and first-episode psychosis at the individual level, Psychological Medicine 43(12), p. 2547-2562, Cambridge University Press, doi:10.1017/S003329171300024X
(2013) Interaction between effects of genes coding for dopamine and glutamate transmission on striatal and parahippocampal function, Human Brain Mapping 34(9), p. 2244-2258, doi:10.1002/hbm.22061
(2012) Dopaminergic genes influence early response to atypical antipsychotics in patients with first presentation of psychosis, Journal of clinical psychopharmacology 32(4), p. 566-569, LWW
(2012) Preliminary report of biological basis of sensitivity to the effects of cannabis on psychosis: AKT1 and DAT1 genotype modulates the effects of $δ$-9-tetrahydrocannabinol on midbrain and striatal function, Molecular psychiatry 17(12), p. 1152-1155, Nature Publishing Group
(2012) Effect of D-amino acid oxidase activator (DAOA; G72) on brain function during verbal fluency, Human brain mapping 33(1), p. 143-153, Wiley Subscription Services, Inc., A Wiley Company Hoboken
(2012) Genetic vulnerability to psychosis and cortical function: epistatic effects between DAAO and G72, Current pharmaceutical design 18(4), p. 510-517, Bentham Science Publishers
(2012) Effect of D-amino acid oxidase activator (DAOA; G72) on brain function during verbal fluency, Human Brain Mapping 33(1), p. 143-153, Wiley Subscription Services, Inc., A Wiley Company Hoboken, doi:10.1002/hbm.21198
(2012) Preliminary report of biological basis of sensitivity to the effects of cannabis on psychosis: AKT1 and DAT1 genotype modulates the effects of δ-9-tetrahydrocannabinol on midbrain and striatal function, Molecular Psychiatry 17(12), p. 1152-1155, Nature Publishing Group, doi:10.1038/mp.2011.187
(2012) Genetic Vulnerability to Psychosis and Cortical Function: Epistatic Effects between DAAO and G72, Current Pharmaceutical Design 18(4), p. 510-517, Bentham Science Publishers, doi:10.2174/138161212799316037
(2012) Dopaminergic genes influence early response to atypical antipsychotics in patients with first presentation of psychosis, Journal of Clinical Psychopharmacology 32(4), p. 566-569, LWW, doi:10.1097/JCP.0b013e31825ec2cb
(2011) No association of Disrupted-in-Schizophrenia-1 variation with prefrontal function in patients with schizophrenia and bipolar disorder, Genes, Brain and Behavior 10(3), p. 276-285, Blackwell Publishing Ltd Oxford, UK
(2011) Pattern of neural responses to verbal fluency shows diagnostic specificity for schizophrenia and bipolar disorder, BMC psychiatry 11, p. 1-10, BioMed Central
(2011) Differential effects of DAAO on regional activation and functional connectivity in schizophrenia, bipolar disorder and controls, Neuroimage 56(4), p. 2283-2291, Elsevier
(2011) Differential effects of DAAO on regional activation and functional connectivity in schizophrenia, bipolar disorder and controls, NeuroImage 56(4), p. 2283-2291, Elsevier, doi:10.1016/j.neuroimage.2011.03.037
(2011) The effects of DISC1 risk variants on brain activation in controls, patients with bipolar disorder and patients with schizophrenia, Psychiatry Research - Neuroimaging 192(1), p. 20-28, Elsevier, doi:10.1016/j.pscychresns.2011.01.015
(2011) No association of Disrupted-in-Schizophrenia-1 variation with prefrontal function in patients with schizophrenia and bipolar disorder, Genes, Brain and Behavior 10(3), p. 276-285, Blackwell Publishing Ltd Oxford, UK, doi:10.1111/j.1601-183X.2010.00665.x
(2011) Pattern of neural responses to verbal fluency shows diagnostic specificity for schizophrenia and bipolar disorder, BMC Psychiatry 11, p. 1-10, BioMed Central, doi:10.1186/1471-244X-11-18
(2010) The 2nd Schizophrenia International Research Society Conference, 10- -14 April 2010, Florence, Italy: Summaries of oral sessions, Schizophrenia research 124(1-3), p. e1--e62, Elsevier
(2010) The 2nd Schizophrenia International Research Society Conference, 10-14 April 2010, Florence, Italy: Summaries of oral sessions, Schizophrenia Research 124(1-3), p. e1--e62, Elsevier, doi:10.1016/j.schres.2010.09.008
(2009) Altered effect of dopamine transporter 3′ UTR VNTR genotype on prefrontal and striatal function in schizophrenia, Archives of general psychiatry 66(11), p. 1162-1172, American Medical Association
(2009) The effect of COMT, BDNF, 5-HTT, NRG1 and DTNBP1 genes on hippocampal and lateral ventricular volume in psychosis, Psychological medicine 39(11), p. 1783-1797, Cambridge University Press
(2009) Epistasis between the DAT 3′ UTR VNTR and the COMT Val158Met SNP on cortical function in healthy subjects and patients with schizophrenia, Proceedings of the National Academy of Sciences 106(32), p. 13600-13605, National Academy of Sciences
(2009) An association study of the neuregulin 1 gene, bipolar affective disorder and psychosis, Psychiatric genetics 19(3), p. 113-116, LWW
(2009) Genetic vulnerability to affective psychopathology in childhood: a combined voxel-based morphometry and functional magnetic resonance imaging study, Biological psychiatry 66(3), p. 231-237, Elsevier
(2009) Opposite Effects of Catechol-O-Methyltransferase Val158Met on Cortical Function in Healthy Subjects and Patients with Schizophrenia, Biological Psychiatry 65(6), p. 473-480, Elsevier, doi:10.1016/j.biopsych.2008.09.027
(2009) Altered effect of dopamine transporter 3′UTR VNTR genotype on prefrontal and striatal function in schizophrenia, Archives of General Psychiatry 66(11), p. 1162-1172, American Medical Association, doi:10.1001/archgenpsychiatry.2009.147
(2009) An association study of the neuregulin 1 gene, bipolar affective disorder and psychosis, Psychiatric Genetics 19(3), p. 113-116, LWW, doi:10.1097/YPG.0b013e32832a4f69
(2009) Increased inferior frontal activation during word generation: A marker of genetic risk for schizophrenia but not bipolar disorder?, Human Brain Mapping 30(10), p. 3287-3298, Wiley Subscription Services, Inc., A Wiley Company Hoboken, doi:10.1002/hbm.20749
(2009) Genetic Vulnerability to Affective Psychopathology in Childhood: A Combined Voxel-Based Morphometry and Functional Magnetic Resonance Imaging Study, Biological Psychiatry 66(3), p. 231-237, Elsevier, doi:10.1016/j.biopsych.2009.01.033
(2009) The effect of COMT, BDNF, 5-HTT, NRG1 and DTNBP1 genes on hippocampal and lateral ventricular volume in psychosis, Psychological Medicine 39(11), p. 1783-1797, Cambridge University Press, doi:10.1017/S0033291709990316
(2008) The effects of neuregulin1 on brain function in controls and patients with schizophrenia and bipolar disorder, Neuroimage 42(2), p. 817-826, Elsevier
(2008) Effect of disrupted-in-schizophrenia-1 on pre-frontal cortical function, Molecular psychiatry 13(10), p. 915-917, Nature Publishing Group
(2008) Effect of disrupted-in-schizophrenia-1 on pre-frontal cortical function, Molecular Psychiatry 13(10), p. 915-917, Nature Publishing Group, doi:10.1038/mp.2008.76
(2008) Association of DAO and G72(DAOA)/G30 genes with bipolar affective disorder, American Journal of Medical Genetics, Part B: Neuropsychiatric Genetics 147(6), p. 914-917, Wiley Subscription Services, Inc., A Wiley Company Hoboken, doi:10.1002/ajmg.b.30682
(2006) Association of the dysbindin gene with bipolar affective disorder, American Journal of Psychiatry 163(9), p. 1636-1638, American Psychiatric Association
(2006) Bipolar 1 disorder is not associated with the RGS4, PRODH, COMT and GRK3 genes, Psychiatric Genetics 16(6), p. 229-230, LWW
(2006) Bipolar 1 disorder is not associated with the RGS4, PRODH, COMT and GRK3 genes, Psychiatric Genetics 16(6), p. 229-230, LWW, doi:10.1097/01.ypg.0000242190.43773.ce
(2006) Association of the dysbindin gene with bipolar affective disorder, American Journal of Psychiatry 163(9), p. 1636-1638, American Psychiatric Association, doi:10.1176/ajp.2006.163.9.1636
Publicações em conferências
(2020) The Use of Consumer Neuroscience Knowledge in Improving Real Promotional Media: The Case of Worten, Smart Innovation, Systems and Technologies 167, p. 202-218, doi:10.1007/978-981-15-1564-4_20
(2009) Epistasis between the DAT 3′ UTR VNTR and the COMT Val158Met SNP on cortical function in healthy subjects and patients with schizophrenia, Proceedings of the National Academy of Sciences of the United States of America 106(32), p. 13600-13605, National Academy of Sciences, doi:10.1073/pnas.0903007106
Capítulos de livros
(2020) The Use of Consumer Neuroscience Knowledge in Improving Real Promotional Media: The Case of Worten, Smart Innovation, Systems and Technologies 167, p. 202-218, url, doi:10.1007/978-981-15-1564-4_20